free fatty acid quantification kit Search Results


free fatty acid quantification assay kit  (Danaher Inc)
94
Danaher Inc free fatty acid quantification assay kit
Free Fatty Acid Quantification Assay Kit, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 94/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/free+fatty+acid+quantification+kit/Free+Fatty+Acid+Assay+Kit+-+Quantification/pm34981106-83-10-16
Average 94 stars, based on 1 article reviews
free fatty acid quantification assay kit - by Bioz Stars, 2026-10
94/100 stars
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free fatty acid quantification kit  (US Biological Life Sciences)
90
US Biological Life Sciences free fatty acid quantification kit
EGCG and EC decreased the DNL pathway in HepG2 cells. Cells were treated with EGCG and EC for 72 h. Control cells were treated with 0.1% DMSO. a The expression of enzymes in the DNL pathway was determined by immunoblotting and b percentages calculated for protein expression are compared with 100% of control. c The histogram shows effects of EGCG and EC decreased the percentage of <t>free</t> <t>fatty</t> <t>acid</t> levels compared with the control. Data are expressed as mean ± SD from at least a triplicate of n = 3, *p < 0.05, denoting significant differences compared with the control
Free Fatty Acid Quantification Kit, supplied by US Biological Life Sciences, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/free+fatty+acid+quantification+kit/free+fatty+acid+quantification+kit/pmc05863485-108-14-19
Average 90 stars, based on 1 article reviews
free fatty acid quantification kit - by Bioz Stars, 2026-10
90/100 stars
  Buy from Supplier
free fatty acid assay kit - quantification  (Advisains)
95
Advisains free fatty acid assay kit - quantification
EGCG and EC decreased the DNL pathway in HepG2 cells. Cells were treated with EGCG and EC for 72 h. Control cells were treated with 0.1% DMSO. a The expression of enzymes in the DNL pathway was determined by immunoblotting and b percentages calculated for protein expression are compared with 100% of control. c The histogram shows effects of EGCG and EC decreased the percentage of <t>free</t> <t>fatty</t> <t>acid</t> levels compared with the control. Data are expressed as mean ± SD from at least a triplicate of n = 3, *p < 0.05, denoting significant differences compared with the control
Free Fatty Acid Assay Kit Quantification, supplied by Advisains, used in various techniques. Bioz Stars score: 95/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/free+fatty+acid+quantification+kit/Free+Fatty+Acid+Assay+Kit+-+Quantification/custom%40ab65341%40pmc06370946__mmc2
Average 95 stars, based on 1 article reviews
free fatty acid assay kit - quantification - by Bioz Stars, 2026-10
95/100 stars
  Buy from Supplier

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EGCG and EC decreased the DNL pathway in HepG2 cells. Cells were treated with EGCG and EC for 72 h. Control cells were treated with 0.1% DMSO. a The expression of enzymes in the DNL pathway was determined by immunoblotting and b percentages calculated for protein expression are compared with 100% of control. c The histogram shows effects of EGCG and EC decreased the percentage of free fatty acid levels compared with the control. Data are expressed as mean ± SD from at least a triplicate of n = 3, *p < 0.05, denoting significant differences compared with the control

Journal: Cancer Cell International

Article Title: Epistructured catechins, EGCG and EC facilitate apoptosis induction through targeting de novo lipogenesis pathway in HepG2 cells

doi: 10.1186/s12935-018-0539-6

Figure Lengend Snippet: EGCG and EC decreased the DNL pathway in HepG2 cells. Cells were treated with EGCG and EC for 72 h. Control cells were treated with 0.1% DMSO. a The expression of enzymes in the DNL pathway was determined by immunoblotting and b percentages calculated for protein expression are compared with 100% of control. c The histogram shows effects of EGCG and EC decreased the percentage of free fatty acid levels compared with the control. Data are expressed as mean ± SD from at least a triplicate of n = 3, *p < 0.05, denoting significant differences compared with the control

Article Snippet: Long chain free fatty acid products of the DNL pathway were evaluated using the Free Fatty Acid Quantification Kit (US, Biological, MA, USA).

Techniques: Control, Expressing, Western Blot

Schematic diagram illustrating the principal mechanism of EGCG and EC in the DNL pathway-mediated apoptosis in HepG2 cells. EGCG and EC decrease the expression of enzymes in DNL pathway including ACC and FASN, which is then results in reducing free fatty acid levels. The possible mechanisms of the anti-DNL pathway of EGCG and EC may be resulted from inhibition of EGCG on the tyrosine kinase receptor mediating PI3K/Akt/mTORC1/SREBP1 axis [ , , ]. Subsequently, reduction of free fatty acid exhibits inhibition of mitochondrial CPT-1 activity, thereby contributing to disruption of lipid β-oxidation . This inhibition results in promoting an increase of ceramide levels. As a result, ceramide can promote mitochondrial dependent apoptosis by activating the interaction of Bcl2 and BNIP3 [ , ]. In addition, accumulation of ROS due to mitochondrial dysfunction and NADPH accumulation contributes to apoptosis in HepG2

Journal: Cancer Cell International

Article Title: Epistructured catechins, EGCG and EC facilitate apoptosis induction through targeting de novo lipogenesis pathway in HepG2 cells

doi: 10.1186/s12935-018-0539-6

Figure Lengend Snippet: Schematic diagram illustrating the principal mechanism of EGCG and EC in the DNL pathway-mediated apoptosis in HepG2 cells. EGCG and EC decrease the expression of enzymes in DNL pathway including ACC and FASN, which is then results in reducing free fatty acid levels. The possible mechanisms of the anti-DNL pathway of EGCG and EC may be resulted from inhibition of EGCG on the tyrosine kinase receptor mediating PI3K/Akt/mTORC1/SREBP1 axis [ , , ]. Subsequently, reduction of free fatty acid exhibits inhibition of mitochondrial CPT-1 activity, thereby contributing to disruption of lipid β-oxidation . This inhibition results in promoting an increase of ceramide levels. As a result, ceramide can promote mitochondrial dependent apoptosis by activating the interaction of Bcl2 and BNIP3 [ , ]. In addition, accumulation of ROS due to mitochondrial dysfunction and NADPH accumulation contributes to apoptosis in HepG2

Article Snippet: Long chain free fatty acid products of the DNL pathway were evaluated using the Free Fatty Acid Quantification Kit (US, Biological, MA, USA).

Techniques: Expressing, Inhibition, Activity Assay, Disruption